Descreve-se um surto de intoxicação por Senna occidentalis em javalis no Estado de Goiás. De um rebanho de 80 javalis, 15 adoeceram e um morreu. Os sinais clínicos observados foram apatia, prostração, ataxia, tremores musculares, incoordenação, relutância em mover-se, decúbito esternal ou lateral, paresia e paralisia espástica, principalmente dos membros pélvicos. As principais alterações macroscópicas consistiam de palidez moderada a acentuada da musculatura esquelética dos membros pélvicos e torácicos e no lombo, especialmente nos músculos da coxa. Microscopicamente, observou-se degeneração e necrose flocular, multifocal, leve a moderada, monofásica, com fragmentação de fibras na musculatura esquelética. Nos cortes transversais, havia fibras musculares tumefeitas e hipereosinofílicas. Adicionalmente, havia degeneração microvacuolar hepatocelular difusa, leve a moderada. A atividade sérica da CK estava acentuadamente elevada em dois javalis avaliados e da TGO aumentou em um javali afetado.

An outbreak of poisoning by Senna occidentalis in wild boars in Goiás, Brazil, is described. Out of 80 wild boars, 15 were affected and one died. Clinical signs included apathy, muscle tremors, incoordination, reluctance to move, sternal or lateral recumbence, and paresis and spastic paralysis, especially in the hind limbs. Gross lesions were characterized by moderate to severe paleness of the skeletal muscles of the hind and thoracic limbs and loin, and were marked in the thigh muscles. Histologically, a monophasic, mild to moderate, multifocal floccular muscle degeneration and necrosis with skeletal fiber sarcoplasmic fragmentation was observed. In transverse sections there were multiple swollen and hypereosinophilic muscle fibers. In addition, mild to moderate, diffuse microvacuolar degeneration was seen in the liver. Creatine phosphokinase serum activity was severely elevated in two evaluated wild boars. In one of them, there was elevation of glutamic oxaloacetic transaminase serum activity.